Abstract
Some microbial metabolites can be immunomodulatory, but there is limited understanding of how these contribute to inter-individual variation in response to infection. In a recent study in Science, Steed et al. (2017) show that the bacterial metabolite desaminotyrosine (DAT) increases type I interferon expression, resulting in an improved immune response to influenza infection. Some microbial metabolites can be immunomodulatory, but there is limited understanding of how these contribute to inter-individual variation in response to infection. In a recent study in Science, Steed et al. (2017) show that the bacterial metabolite desaminotyrosine (DAT) increases type I interferon expression, resulting in an improved immune response to influenza infection.
Original language | English |
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Pages (from-to) | 400-402 |
Number of pages | 3 |
Journal | Immunity |
Volume | 47 |
Issue number | 3 |
DOIs | |
State | Published - 19 Sep 2017 |
Externally published | Yes |
Bibliographical note
Publisher Copyright:© 2017 Elsevier Inc.