Rapid chemical determination of chloramphenicol acetylation by cell extracts and by cell suspensions of E. coli B

N. Garber; D. Zohar; H. Michlin

doi:10.1038/219401a0

Rapid chemical determination of chloramphenicol acetylation by cell extracts and by cell suspensions of E. coli B

N. Garber, D. Zohar, H. Michlin

The Mina and Everard Goodman Faculty of Life Sciences - at Bar-Ilan University

Bar-Ilan University

Research output: Contribution to journal › Letter › peer-review

2 Scopus citations

Abstract

STUDIES of subcellular systems of resistant E. coli K-12 (ref. 1) and staphylococci² have proved that the basis of chloramphenicol resistance in these bacteria is their ability to acetylate the drug. These results agreed with Miyamura's report³ on the relationship between the resistance to chloramphenicol of naturally isolated enterobacteria carrying R-factor and of staphylococci and their ability to inactivate the drug.

Original language	English
Pages (from-to)	401-402
Number of pages	2
Journal	Nature
Volume	219
Issue number	5152
DOIs	https://doi.org/10.1038/219401a0
State	Published - 1968

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title = "Rapid chemical determination of chloramphenicol acetylation by cell extracts and by cell suspensions of E. coli B",

abstract = "STUDIES of subcellular systems of resistant E. coli K-12 (ref. 1) and staphylococci2 have proved that the basis of chloramphenicol resistance in these bacteria is their ability to acetylate the drug. These results agreed with Miyamura's report3 on the relationship between the resistance to chloramphenicol of naturally isolated enterobacteria carrying R-factor and of staphylococci and their ability to inactivate the drug.",

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N2 - STUDIES of subcellular systems of resistant E. coli K-12 (ref. 1) and staphylococci2 have proved that the basis of chloramphenicol resistance in these bacteria is their ability to acetylate the drug. These results agreed with Miyamura's report3 on the relationship between the resistance to chloramphenicol of naturally isolated enterobacteria carrying R-factor and of staphylococci and their ability to inactivate the drug.

AB - STUDIES of subcellular systems of resistant E. coli K-12 (ref. 1) and staphylococci2 have proved that the basis of chloramphenicol resistance in these bacteria is their ability to acetylate the drug. These results agreed with Miyamura's report3 on the relationship between the resistance to chloramphenicol of naturally isolated enterobacteria carrying R-factor and of staphylococci and their ability to inactivate the drug.

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Rapid chemical determination of chloramphenicol acetylation by cell extracts and by cell suspensions of E. coli B

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