Early brain atrophy in persistent hyperinsulinemic hypoglycemia of infancy

Zohar Nachum; Izhar Ben-Shlomo; Yardena Rakover; Ehud Weiner; Eliezer Shalev

doi:10.1067/mpd.2002.128546

Early brain atrophy in persistent hyperinsulinemic hypoglycemia of infancy

Zohar Nachum
, Izhar Ben-Shlomo
, Yardena Rakover
, Ehud Weiner
, Eliezer Shalev

Emek Medical Center
Technion-Israel Institute of Technology

Research output: Contribution to journal › Article › peer-review

3 Scopus citations

Abstract

We present two siblings with persistent hyperinsulinemic hypoglycemia of infancy, accelerated intrauterine growth and early neonata brain atrophy. Fetal plasma glucose and insulin levels in the second sibling revealed normoglycemia despite hyperinsulinemia. The absence of intrauterine hypoglycemia suggests that the brain damage is not secondary to hypoglycemia and other etiologies must be considered.

Original language	English
Pages (from-to)	706-709
Number of pages	4
Journal	Journal of Pediatrics
Volume	141
Issue number	5
DOIs	https://doi.org/10.1067/mpd.2002.128546
State	Published - 1 Nov 2002
Externally published	Yes

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10.1067/mpd.2002.128546

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title = "Early brain atrophy in persistent hyperinsulinemic hypoglycemia of infancy",

abstract = "We present two siblings with persistent hyperinsulinemic hypoglycemia of infancy, accelerated intrauterine growth and early neonata brain atrophy. Fetal plasma glucose and insulin levels in the second sibling revealed normoglycemia despite hyperinsulinemia. The absence of intrauterine hypoglycemia suggests that the brain damage is not secondary to hypoglycemia and other etiologies must be considered.",

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AU - Ben-Shlomo, Izhar

AU - Rakover, Yardena

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AU - Shalev, Eliezer

PY - 2002/11/1

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N2 - We present two siblings with persistent hyperinsulinemic hypoglycemia of infancy, accelerated intrauterine growth and early neonata brain atrophy. Fetal plasma glucose and insulin levels in the second sibling revealed normoglycemia despite hyperinsulinemia. The absence of intrauterine hypoglycemia suggests that the brain damage is not secondary to hypoglycemia and other etiologies must be considered.

AB - We present two siblings with persistent hyperinsulinemic hypoglycemia of infancy, accelerated intrauterine growth and early neonata brain atrophy. Fetal plasma glucose and insulin levels in the second sibling revealed normoglycemia despite hyperinsulinemia. The absence of intrauterine hypoglycemia suggests that the brain damage is not secondary to hypoglycemia and other etiologies must be considered.

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Early brain atrophy in persistent hyperinsulinemic hypoglycemia of infancy

Abstract

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