| Original language | English |
|---|---|
| Pages (from-to) | 2390-2392 |
| Number of pages | 3 |
| Journal | Neural Regeneration Research |
| Volume | 16 |
| Issue number | 12 |
| DOIs | |
| State | Published - Dec 2021 |
| Externally published | Yes |
Funding
Heneka MT, Kummer MP, Stutz A, Delekate A, Schwartz S, Vieira-Saecker A, Griep A, Axt D, Remus A, Tzeng TC, Gelpi E, Halle A, Korte M, Latz E, Golenbock DT 縃缀 N?R? is activated in Alzheimer ? s disease and contributes to pathology in APP/PS1 mice. Nature 493:674-678. Israelov H, Ravid O, Atrakchi D, Rand D, Elhaik S, Bresler Y唀 Twitto 爀✁?eenberg R 唀 Omesi ?唀 ?iraz 爀?altsman S 唀 Gosselet F, Schnaider Beeri M, Cooper I (2020) Caspase 爃 has a critical role in blood 爁༁?ain barrier injury and its inhibition contributes to multifaceted repair堀 J Neuroinflammation 圃堀 This work was supported by the Defense Jang Y, Lee AY, Jeong SH, Park KH, Paik MK, Cho NJ, Threat Reduction Agency 爀㨁紁崁?t Science 頀 Kim JE, Cho MH (2015) Chlorpyrifos induces NLRP3 Technology Office for Chemical 頀 Biological mitochondrial oxidative stress in human keratinocyte inflammasome and pyroptosis ?apoptosis via At least in our experience, targeting Defense 縀䔁紃堀 缃唀 and tHhaeC aeTh ceemlls.i aTo xicology 338:37-46. Rubin Excellence in Biomedical Research ? Keaney J, Campbell M (2015) The dynamic blood-brain molecules which are downstream 縀⼀㸃爃球唀 ?? ? barrier. FEBS J 282:4067-4079. 18, IL-8) or upstream (NLRP3) to caspase-1 The TELEM Program supported by the Aaron ?amkanfi M?KannegantiTD?VanDamme ??VandenBerghe
| Funders | Funder number |
|---|---|
| Defense Jang Y | |
| Threat Reduction Agency |